Expert Rev Clin Immunol. We use a stratified with randomization with help block diet. Patients are recruited from all over Germany. Witte M. Later, all data will be digitized and stored in a central can. First, we include patients with any MS medication or no medication. J Keto. Mult Scler Relat Disord. Demyelination increases axonal stationary mitochondrial size and the speed of axonal mitochondrial transport.
Multiple sclerosis is a condition that affects the nervous system, and some people believe that a ketogenic diet may slow the progression of the disease or control its symptoms. The ketogenic diet is low in carbohydrate, moderate in protein, and high in fat. There is currently not enough evidence to recommend it for everyone with multiple sclerosis. Proponents of the ketogenic diet believe that it can help people lose weight and possibly reverse degenerative diseases. The ketogenic diet does offer some benefits, but people with multiple sclerosis MS need to consider certain factors before trying the diet. The purpose of the ketogenic diet is to encourage the body to switch from burning carbohydrates to burning fats. The body usually turns the carbohydrates from food into the sugar glucose, which it then uses for energy or stores for later. However, if a person starves their body of glucose by removing carbohydrates from their diet, the body will switch to using fats as its source of fuel instead. People refer to this adaptation as ketosis, which gives the diet its name.
Second, a dietary intervention study cannot be blinded completely. While I didn’t have a resource like MS Mindshift all those years ago, it was through online communities and my own research not a DVD and pamphlet that I learned how much of an impact things like diet and exercise can have when it comes to managing MS. The secondary MRI endpoint is brain atrophy determined by percentage brain volume change. This app provides access to expert content on MS, as well as peer support through one-on-one conversations and live group discussions. These changes may confer resistance to glucose entry into demyelinated axons, depriving them of adequate fuel supply resulting in glucose hypometabolism [ 59 ]. Nature, nurture, and microbes: the development of multiple sclerosis.